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In the actual nucleus involving HIV-infected regenerating cellular material, NF-��B p50/p50 homodimers likely will your 5��LTR. The particular p50/p50 homodimer falls short of the main transactivation site that is certainly seen in the p50/RelA heterodimer (your prototypical NF-��B sophisticated). Moreover, p50 homodimers can definitely hinder transcription by enrolling HDAC-1 to the LTR along wit

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