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Consequently, it absolutely was reasonable that the mechanism of inhibition of key WT1 isoform-mediated transcriptional activation of Bcl-xL and Bcl-2 by Ex4a(+)WT1 isoform may very well be opposition with important WT1 isoform for interacting using the transcriptional co-activators other than WT1 alone, resulting inside the abrogation of big WT1 isoform-mediated transcriptional action in leukemia

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