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For that reason, it had been acceptable the mechanism of inhibition of main WT1 isoform-mediated transcriptional activation of Bcl-xL and Bcl-2 by Ex4a(+)WT1 isoform could be opposition with main WT1 isoform for interacting while using the transcriptional co-activators other than WT1 itself, ensuing in the abrogation of important WT1 isoform-mediated transcriptional activity in leukemia cells. Non

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